Cataract surgery xalatan explain has become ubiquitous in the management of glaucoma patients as the basis for writing his article
Cataract surgery xalatan
We need to know that cataracts are a major cause of blindness in the world. Cataract is an aging-related vision disorder that causes vision to become cloudy and cloudy cloudy. Cataracts make you look like behind a thick dusty window. However, many people are reluctant to undergo surgery because they fear the risk of complications. Cataracts appear in the lens of the eye, a transparent colored crystal structure that is just behind the pupil. This one eye structure works like a camera lens by focusing light on the retina of the eye. The lens also adjusts the focus of the eye, which makes it possible to see things clearly both near and far.
Cataracts and glaucoma are two types of eye diseases that can both cause blindness. Glaucoma is a type of visual impairment with damage to the optic nerve caused by pressure in the eye. Glaucoma is a visual disease due to high pressure on the nerve fluid that damages the eyes. Glaucoma is characterized when the optic nerve is damaged due to pressure on the eye. The cause of glaucoma is an increase in pressure in the eye (intraocular pressure), both due to excessive production of eye fluid, as well as due to obstruction of the drainage of the fluid. This pressure can damage the retinal nerve fibers and the optic nerve that connects the eye to the brain as well. Until now, it was not clear why the production of eye fluids could be excessive or why the drainage can be blocked.
In the mid-1990s with the emergence of latanoprost (Xalatan), it has become ubiquitous in the management of glaucoma patients. Many case reports show a relationship between the incidence of CME and the use of latanoprost. CME (cystoid macular edema) is a relatively feared complication. Many cataract patients enjoy immediate postoperative improvement in visual acuity caused by complications such as CME. The relationship between latanoprost and CME in the study period seems very low. CME occurs in only three high-risk patients who experience posterior capsule rupture at the time of cataract removal. Overall, the incidence of CME with latanoprost seems uncommon. However, CME is at high risk in patients who have complicated cataract surgery. In a separate study evaluating latanoprost use in conjunction with non-steroidal eyedrops, latanoprost succeeded in reducing intraocular pressure (IOP) while side-effects were minimized. Latanoprost has been linked to disorders of blood watery barriers. However, using latanoprost after cataract surgery should only be careful and tell them that if they experience a decrease in visual acuity, they should return to the doctor.
The authors conclude that the effects associated with latanoprost can be prevented if nonsteroidal eyedrops are given simultaneously. The use of latanoprost has never been described as a disturbance to blood-water and blood retinal barriers in an animal or human eye or causes CME in pseudophakias which has been prolonged. This shows that when the eye is compressed by surgery, the lens epithelial cells proliferate and transform, and various chemical mediators are created. The process of changing epithelial cells is believed to modify postoperative inflammation, resulting in the development of the fibrin pupil membrane.
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As reference Cataract surgery xalatan please read on Wikipedia about the Latanoprost